Preliminary scientific studies show conolidine might inhibit unique ion channels, minimizing neuronal excitability and limiting discomfort alerts. This mechanism is especially related in neuropathic ache, where by irregular signaling results in persistent pain. Also, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to discomfort notion. Computational do... https://robertm258qmg6.wikifrontier.com/user