Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Alternatively, our ability to rescue SERT perform in KI midbrain synaptoneurosomes via the inhibition of FAK implies elevated FAK signaling downstream on the Pro32Pro33 mutant, as confirmed by enhanced pFAK localization in 5-HT synapses. Our https://bobbyl531pal3.wikimillions.com/user